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Covid vaccines may cause diseases in ’10 to 15 years,’ according to an MIT scientist.

WATCH: Covid Vaccines May Cause Diseases In '10 To 15 Years,' According To An MIT Scientist

“Down the line, we’re in for a tremendous surprise,” Dr. Stephanie Seneff predicts.

The RAIR Foundation USA is pleased to share an exclusive conversation with renowned scientist Stephanie Seneff on the impact of mRNA vaccinations. RAIR also features Physiology and Biology Professor Madeline Weld, Ph.D., who has graciously used her knowledge to write about the interview for RAIR’s readers (scroll down).

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Dr. Seneff is the author of Toxic Legacy: How the Weedkiller Glyphosate Is Destroying Our Health and the Environment, which was published by MIT’s Computer Science and Artificial Intelligence Laboratory.

“Worse Than the Disease?” wrote the MIT scientist alongside Dr. Greg Nigh in a research paper titled “Worse Than the Disease?” For the International Journal of Vaccine Theory, Practice, and Research, I reviewed “Some Possible Unintended Consequences of the mRNA Vaccines Against COVID-19.”

Dr. Weld elucidates:

“Dr. Seneff’s background caused her to develop reservations about the widely marketed Covid-19 vaccination. These vaccinations are made with a new mRNA technique, and their long-term consequences are uncertain.

However, there is enough available scientific evidence to explain some of the reasons behind the current detrimental effects of these vaccines, as well as what they may cause in the coming years and decades. She expects a rise in autoimmune and neurological illnesses, which can take 10 to 15 years to manifest.”

The following are some of the key points from the RAIR exclusive interview:

Dr. Seneff “expects long-term consequences that will not be immediately attributed to the vaccine.” Increases in auto-immune and neurological illnesses, which can take 10 to 15 years to manifest.” “Down the line, we’re in for a tremendous surprise,” she predicts.
Dr. Seneff feels that the vaccine will aggravate the symptoms of Parkinson’s patients.
Those who asserted that mRNA has no effect on DNA are “wrong.”
Spike protein “has really become the most dangerous element of the virus” and continues to remain even after Covid is no longer present.

“An increase in Creutzfeldt-Jacob disease (CJD), a prion disease (or protein misfolding disease) similar to mad cow disease, is one of the possibilities she sees.”
“Alzheimer’s disease is on the rise, and people are developing it at a younger and younger age. This trend will be aided by the vaccination rollout’s hasty and haphazard implementation.”
Watch the video and read on for more information:

Seneff has been researching glyphosate and has written a new book about it (Toxic Legacy: How the Weedkiller Glyphosate is destroying our health and the environment). She’s been studying this subject for about a decade. She believes that glyphosate poisons the immune system, causing immunological weakness and autoimmune disease, which increases Covid-19’s impact in terms of contributing to the cytokine storm, lungs swelling with fluid, bleeding, and thrombosis. Glyphosate and the immune system are covered in depth in her book.

This work was thoroughly scrutinized (6 reviewers). They had heard about the potential that the spike protein was a prion, but decided against including it in their manuscript since it was too speculative.

One of the reviewers, though, advised that they include a debate about it, prompting them to deepen their investigation and discover that it connected a lot of dots. Prion-like proteins are found in several viruses. She discovered about an organic farmer in the United Kingdom who linked mad cow disease to magnesium toxicity caused by a pesticide applied to cows’ backs. Because hazardous chemicals cause proteins to misfold, Seneff argues that prion disorders are linked to them.

Prion proteins can cause misfolding in unrelated proteins. There is a list of proteins that will “catch the disease” if they come into contact with a prion or prion-like protein that misfolds in a specific fashion known as “beta-sheets.” It causes other proteins in the same cell to misfold and clump together, forming fibrils that finally precipitate out. Alzheimer’s disease is an example of a prion-like disease. Alzheimer’s disease is linked to amyloid beta plaque. Amyloid beta plaques generally fold into alpha helices and act as screws in the membrane. This is a feature shared by all prion proteins. They generally form an alpha helix when they enter a membrane.

When prion proteins misfold, which can happen when there are too many of them in the cytoplasm, such as when there is too much alpha-synuclein amyloid-beta, the alpha-synuclein amyloid-beta molecules start glomming together in these soluble beta sheets, resulting in soluble complexes of beta sheets containing many amyloid betas.

Misfolding of alpha-synuclein is linked to Parkinson’s disease.
Amyloid-beta is linked to Alzheimer’s disease, while TDP43 is linked to ALS.

There is a typical prion protein that causes mad cow disease in cows and CJD in humans. So you have individual proteins connected to certain neurodegenerative illnesses, and each of those proteins behaves in the same way, with the alpha helix transforming into a beta sheet that becomes soluble in the cytoplasm and creating problems. Many different debilitating diseases follow this trend. This is caused by a prion-like protein that is frequently introduced from the outside.

One example is Parkinson’s disease. You can start with a prion-like protein generated by E. coli. If it becomes too much, it begins to misfold and develop prion C in the gut. The immune cells then take up the protein and transport it to the spleen.

In the spleen, there are germinal centers where the prion proteins really begin rolling. It appears to be a unique factory dedicated to dealing with prion proteins and eliminating them. Immune cells in the spleen’s germinal centers package prion proteins into little particles called exosomes, which they release.

Exosomes are crucial to the disease process that these vaccinations are expected to trigger. Parkinson’s disease begins with germinal centers in the spleen releasing exosomes containing a dangerously misfolded protein. The cells are attempting to eliminate and disperse them. You could cough them up, release them through your skin, and they could end up in your breast milk. They are exported as these tiny exosomes in order to get rid of them and shed them. They are also delivered to the brain stem via the vagus nerve. And this is how Parkinson’s disease develops. Parkinson’s disease begins in the gut, then progresses to the spleen, the vagus nerve, and finally the brain.

Source
www.nature.comwww.hsph.harvard.eduwww.rairfoundation.com

Margaret Taylor

Experienced communications professional with 10 years of experience in international journalism.

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